module 8: Spinal cord injuries, Autonomic Dysreflexia, Back pain, Osteomyelitis, & Fractures

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Spinal cord injury

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Spinal cord injury

An injury to the spinal cord, vertebral column, supporting soft tissue, or intervertebral disks caused by trauma

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Pathophysiology of SCI

  • Ranges from transient concussion (which is fully recoverable) to contusion, laceration, and compression of the spinal cord tissue to complete severing of the spinal cord

    • Renders patient paralyzed below the level of injury

  • Vertebra C5-C7, T12, and L1 are the most susceptible to injury due to a greater range of motion

  • Primary injuries = result of initial insult or trauma; permanent damage

  • Secondary injuries = result from hypoxemia, edema, or hemorrhage which damages the tissue

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Risk factors of SCI

  • Younger age

  • Male gender

  • Alcohol and illicit drug use

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clinical manifestations of SCI

  • Complete spinal cord lesion = loss of sensory and voluntary motor communication; paraplegia or tetraplegia

    • Incomplete spinal cord lesion = sensory and motor communication is preserved below the lesion

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s/s of SCI

  • Total or partial paralysis

  • Loss of bowel and bladder control

  • Loss of sweating

  • Reduction in BP with loss of peripheral vascular resistance

  • Acute back or neck pain

  • Respiratory dysfunction (depending on level of injury)

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Associated priorities of SCI

  • Immobilization in the extended position until proper assessment and treatment is performed

    • Incomplete injury may become complete if pt continues to move after trauma event

  • Neurological assessment

  • X-ray

  • CT scan

  • MRI scan for  further workup

  • Monitor respirations/pattern

  • Vitals

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Usual treatment of SCI

  • Respiratory therapy

  • Pacing

  • High dose IV corticosteroids

    • Methylprednisolone

  • Immobilization

    • Stabilization of vertebral column

  • Reduction of dislocations

  • Skeletal traction (cervical injuries)

  • Surgical intervention (thoracic and lumbar injuries)

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Nursing considerations of SCI

  • Spinal shock

  • Neurogenic shock

  • VTE

    • DVT

    • PE

  • Pressure injuries

  • Monitor respirations and breathing pattern

  • Lung sounds and cough

  • Monitor for changes in motor or sensory function; report immediately

  • Assess for spinal shock

  • Monitor for bladder retention or distention, gastric dilation, and ileus

  • Temperature; potential hyperthermia

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Characteristics of central cord syndrome

  • Motor deficits

    • In the upper extremities compared to the lower extremities

    • Sensory loss more pronounced in upper extremities

  • bowel/bladder dysfunction

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Cause of central cord syndrome

  • Injury

  • Edema

  • Hyperextension injuries

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Characteristics of anterior cord syndrome

  • Loss of pain (below the level of lesion)

  • Loss of temperature function (below the level of lesion)

  • Loss of motor function (below the level of lesion)

  • Light touch, position, and vibration sensation remain intact

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Cause of anterior cord syndrome

  • Acute disc herniation

  • Hyperflexion injuries associated with fracture or dislocation of vertebra

  • Injury to anterior spinal artery

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Characteristics of lateral cord syndrome

  • Ipsilateral paralysis or paresis is noted

  • Ipsilateral loss of touch, pressure, and vibration

  • Contralateral loss of pain and temperature

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Cause of lateral cord syndrome

  • Lesion is caused by a vertical transection

  • Result of knife or missile injury, fracture/dislocation, or acute ruptured disk

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Skeletal traction

  • Used to reduce cervical fractures and align the cervical spine

  • Traction is applied to via weights

  • As the traction increases, the spaces between the intervertebral disks widen, allowing the disks to slip back into place

  • Reduction in traction occurs after proper alignment is restored

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Spinal shock

Sudden depression of reflex activity below the level of injury caused by muscles being paralyzed and flaccid

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s/s of spinal shock

  • Decreased BP

  • Bradycardia

  • Bowel distention

  • Paralytic ileus

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Considerations of spinal shock

  • Maintain MAP of at least 85 mm Hg

  • NG tube

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Neurogenic shock

  • Result of the loss of autonomic NS function below level of injury

  • Vital organs are affected

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s/s of neurogenic shock

  • Decrease BP, HR, and cardiac output

  • Venous pooling

  • Respiratory complications

    • Decreases vital capacity

    • Retention of secretions

    • Increased PaCO2

    • Decreased O2

    • Pulmonary edema

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Autonomic hyperreflexia

Acute life threatening emergency that occurs as a result of exaggerated autonomic responses to stimuli after spinal shock has been resolved

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Pathophysiology of autonomic dysreflexia

  • Exaggerated autonomic responses to stimuli

  • Triggers:

    • Distended bladder

    • Distention or contraction of visceral organs

      • Bowel

    • Stimulation of the skin

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Risk factors of autonomic dysreflexia

  • SCI

  • Spinal shock

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Clinical manifestations of autonomic dysreflexia

  • Paroxysmal HTN

  • Profuse diaphoresis above injury

  • bradycardia

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s/s of autonomic dysreflexia

  • Severe pounding headache

  • Nausea

  • Nasal congestion

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Associated priorities of autonomic dysreflexia

  • Remove the triggering stimulus

  • Immediately place in sitting position to lower BP

  • Empty bladder

  • Examine for fecal mass and remove

  • Skin assessment for pressure or broken skin

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Usual treatment of autonomic dysreflexia

  • Antihypertensives

    • Slow IV

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Low back pain

  • Caused by musculoskeletal problems such as

    • Acute lumbosacral strain

    • Insatiable lumbosacral ligaments

    • Weak muscles

    • Unequal leg length

    • Intervetebral disc problems

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Pathophysiology of low back pain

  • The spinal column has rigid units = vertebrae and flexible units = intervetebral discs, held together by joints, ligaments, and muscles

  • Spinal curves absorb vertical shocks from movement

  • Abdominal and thoracic muscles are important in lifting and minimize stress on the spinal units

  • Intervertebral discs change with age

    • Fibrocartilage becomes dense and irregularly shaped with time; disc degeneration

  • Low lumbar discs, L4 and L5-S1, are subject tot he greatest mechanical stress and degeneration

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Cauda equina syndrome

compression of a bundle of spinal nerves from the lower spinal cord

  • Results from the compression of the cauda equina, a bundle of spinal nerves from the lower portion of the spinal cord

  • Requires immediate attention before nerve damage occurs

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Risk factors of low back pain

  • Depression

  • Smoking

  • Alcohol abuse

  • Obesity

  • Stress

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Clinical manifestations of low back pain

  • Acute back pain (lasting <3 months)

  • Chronic back pain (lasting 3< without improvement)

  • Fatigue

  • Pain radiating down the leg

    • Radiculopathy = pain from a diseased spinal nerve root

    • Sciatica = pain from inflamed sciatic nerve

  • Affected gait, spinal mobility, reflexes, leg length, motor strength, and sensory perception

  • Cauda equina

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Associated priorities of low back pain

  • Focused history and physical exam

    • Gait evaluation

    • Neurologic testing

  • X-ray, CT scan, MRI, etc.

  • Assess posture, position changes, and gait

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Usual treatment of low back pain

  • Analgesics, rest, and avoidance of strain

  • Activity modification

  • NSAIDs and short term muscle relaxants

  • Opioids (1-2 weeks)

  • Thermal applications

  • Spinal manipulation (chiropractor)

  • Cognitive behavioral therapy

  • Exercise regiments

  • Spinal manipulation

  • PT

  • Acupuncture

  • Massage

  • Avoid stress to back

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Nursing considerations of low back pain

  • Positioning

  • Location, severity, duration, characteristics, radiation (radiculopathy), leg weakness

  • How the pain occurred and has been managed by the patient

  • Work and recreational activities

  • Spinal curvature, back and limb symmetry

  • Palpate paraspinal muscles

  • Movement ability and effects on ADLs

  • DTRs, sensation, and muscle strength

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s/s of cauda equina

  • Sever or progressive neurologic deficit

  • Recent bowel or bladder dysfunction

  • Saddle anesthesia

    • Paresthesias in the but and inner thighs

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Osteomyelitis

  • An infection of the bone that results in inflammation, necrosis, and formation of new bone

  • Can be

    • Hematogenous (bloodborne spread)

    • Contagious-focus (contamination from surgery, fracture, or trauma)

    • With vascular insufficiency

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Pathophysiology of Osteomyelitis

  • Infections caused by staph aureus, MRSA, streptococci, enterococci, or pseudomonas

  • Initial response is inflammation, increased vascularity, and edema; after 2-3 days, thrombosis of local blood vessel occurs causing ischemia with bone necrosis

  • Infection spreads to tissues and joints, and may cause a bone abscess

    • Abscess contains dead bone tissue and cannot heal, but instead new bone growth forms around the dead bone tissue

    • Chronic infection of the bone tissue = chronic osteomyelitis

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Risk factors of Osteomyelitis

  • Old age

  • Poor nourishment

  • Obesity

  • Impaired immune function

  • Chronic illness

  • Long term corticosteroid therapy

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Clinical manifestations of Osteomyelitis

  • Sudden onset of sepsis manifestations

    • Chills

    • High fever

    • Rapid pulse

    • General malaise

  • Infected area becomes painful, swollen, and extremely tender

  • Constant, pulsating pain that intensifies with movement

  • Pus

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Associated priorities of Osteomyelitis

  • Early x-ray

  • MRI

  • Blood studies

  • Bone biopsies

  • Ability to adhere to prescribed therapeutic regimen— antibiotic therapy

  • Signs and symptoms of infection, localized pain, edema, erythema, fever, drainage

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Usual treatment of Osteomyelitis

  • IV antibiotics

  • Prophylactic antibiotics

  • Surgical debridement

    • All dead, infected bone, tissue, and cartilage must be removed for permenant healing

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Nursing considerations of Osteomyelitis

  • Disposable medical tools (to minimize possibility of infection)

  • Encourage adequate hydration, vitamins, and protein

  • Relieving pain

    • Immobilization

    • Elevation

    • Handle with great care and gentleness

    • Administer prescribed analgesics

  • Improving physical mobility

    • Activity is restricted

    • Gentle ROM to joints above and below the affected part

    • Participation in ADLs within limitations

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Contusion

  • soft tissue injury produced by blunt force

    • Pain, swelling, and discoloration: ecchymosis

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Strain

  • Pulled muscle injury to the musculotendinous unit

    • Pain, edema, muscle spasm, ecchymosis, and loss of function are on a continuum graded first, second, and third degree

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Sprain

  • injury to ligaments and supporting muscle fiber around a joint

    • Pain (may increase with motion), edema, tenderness; severity graded according to ligament damage and joint stability

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Dislocation

  • articular surfaces of the joint are not in contact

    • A traumatic dislocation is an emergency with pain change in contour, axis, and length of the limb and loss of mobility

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Subluxation

  • partial or incomplete dislocation

    • Does not cause as much deformity as a complete dislocation

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RICE

  • Rest

  • Ice

  • Compression

  • Elevation

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Fracture

  • A complete or incomplete disruption in the continuity of bone structure and is defined according to its type and extent

  • Occur when a bone is subjected to stress greater than it can absorb

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Pathophysiology of Fractures

May be caused by direct blows, crushing forces, sudden twisting motions, and extreme muscle contractions

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Clinical manifestations of Fractures

  • Adjacent structures may have soft tissue edema, hemorrhage into the muscles and joints, joint dislocations, ruptured tendons, severed nerves, and damaged blood vessels

  • Continuous pain that increases in severity

    • Numbness immediately after

  • Loss of function

  • Displacement, angulation, or rotation

  • Shortening

  • Crepitus

  • Localized edema and echymosis

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Associated priorities of Fractures

  • Immobilization of body part

  • X-ray

  • Adequate splinting

    • Bandaging affected to unaffected extremity or chest

    • Sling

  • Assessment of perfusion and nerve function

  • Cover wound with sterile dressing (open fracture)

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Usual treatment of Fractures

  • Reduction = restoration of bone fragments to anatomic realignment and positioning with realignment

    • Closed uses manipulation and manual traction

    • Open uses surgery and internal fixation devices

  • Immobilization until union occurs

  • IV antibiotics and debridement (open)

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Nursing considerations of Fractures

  • Maintaining and restoring function

    • Position changes

    • Pain relief

    • Isometric exercise

    • Reassurment

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Factors affecting fracture healing

  • Age >40 years

  • Avascular necrosis

  • Bone loss

  • Cigarette smoking

  • Comorbidities (e.g., diabetes, rheumatoid arthritis)

  • Corticosteroids, nonsteroidal anti-inflammatory drugs

  • Extensive local trauma

  • Inadequate immobilization

  • Infection

  • Local malignancy

  • Malalignment of the fracture fragments

  • Space or tissue between bone fragments

  • Weight bearing prior to approval

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Early complications of fractures

  • Shock

  • Fat embolism

  • Acute compartment syndrome

  • DVT; PE

  • Infection

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Late complications of fractures

  • Union

  • Malunion

  • Nonunion

  • AVN of bone

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Types of fractures

  • Closed

  • Open

    • Type I

    • Type II

    • Type III

  • Intra-articular

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Closed fracture

  • Simple fracture

  • Fracture that does not break the skin

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Open fracture

  • Compound or complex fracture

  • Fracture that breaks the skin or mucous membranes around it

  • Type I = clean wound less than 1cm ina simple fracture pattern

  • Type II = larger wound with minimal soft tissue damage; no flaps or avulsions

  • Type III = most severe, highly contaminated, and has extensive soft tissue damage

    • Vascular injury or traumatic amputation

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Intra-articular

Extends into the joint surface of a bone

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Casts

  • Ridgid external immobilizing device that is molded to the contours of the body; must fir the shap of the injured limb to provide the best support

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Cast application

  • Before application

    • General health assessment

    • Emotional status

    • Presenting signs and symptoms and condition of the area (prior to doing immobilzation)

  • Monitoring of neurovascular status and for potential complications

  • Treat lacerations and abrasions before cast, brace, splint

  • Provide information about the purpose of treatment

  • Prepare patient for application by explaining procedure

    • prepare them for what they might feel while being splinted or casted.

    • With the cast- It may feel weird because it is warm as you put it on and gets warmer as it dried. Then it gets hard, this may make them feel claustrophobic

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The 5 P’s of casts

  • Assessing for neurovascular changes using “5 Ps”

    • Pain

    • Pallor

    • Pulselessness

    • Paresthesia

    • Paralysis

  • Monitoring and treating pain

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